Metribolone (methyltrienolone also known as R1881) is a potent, non-aromatizable androgen that is structurally similar to trenbolone and has been called a “mouth sled”. Methyltrienolone binds strongly to the androgen receptor (AR) and is a more potent androgen receptor agonist (activator) than DHT. 17a-Methyltrienolone is listed as 30,000 times more anabolic than methyltestosterone according to Julius Vida in “Androgens and Anabolic Agents: Chemistry and Pharmacology”. Effective doses start at just 25mcg.
Steroid experts William Llewellyn and Patrick Arnold called methyltrienolone one of the “most powerful” anabolic steroids ever made. It is also one of the most hepatotoxic androgens ever produced. Originally developed by Russell-UCLAF in the 1960s, Metribolone’s liver toxicity prevented commercial release. Bill Roberts compared the toxicity of methyltrienolone to taking high doses of Anadrol in combination with high doses of halotestin at the same time.
Dosage: individual course
Doses may range from 0.5 mg to 2 mg per day. day. For the first time, it is not recommended to receive 0.5 mg injections due to its strong androgenic effect. The dosage is strictly individual, but if you have never tried this medicine for the first time, do not take more than 0.5 mg, which is equivalent to 1/4 ml of the solution, and inject very slowly into the muscle.
Maximum shelf life of the drug is not more than 4 weeks.
You may be advised to use liver cleansing supplements, such as Liver Stable, Liv-52 and Essentiale Forte, in the course.
Caution: Those who want to use this medication should take it seriously. This drug is not for steroid beginners.
Methyltrinolone was first described in 1965. It was identified as an extremely potent anabolic drug, far more potent than any commercially available drug at the time. However, despite its relatively high potency, methyltrinolone has a very short history of use. In the late 1960s and early 1970s, the drug was used in clinical practice, primarily for the treatment of advanced breast cancer. Due to the extremely strong anabolic / androgenic ratio, the drug is able to resist the local effects of endogenous estrogens, making it effective in delaying or even regressing tumor growth.